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Shvoong Home>Social Sciences>The ability of mecobalamin to protect the rat optic nerve from injury by crushing Summary

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The ability of mecobalamin to protect the rat optic nerve from injury by crushing

Article Summary by: TsingHua     

Original Author: Journal of Optometry & Ophthalmology
This abstract was translated from 甲钴胺对钳夹伤大鼠视神经的保护作用
Objective:To evaluate the effect of mecobalamin in protecting the optic nerve from injury in experimental animals.Methods:The
rat optic nerve was crushed,then rats were randomly divided into 7 groups:the one-month control group and two-month control group did not receive therapy. The Vit B 12-treated group and mecobalamin-treated group were treated with 10 μg Vit B 12 and mecobalamin,respectively. These groups received intramuscular injections every two days for one month. The mecobalamin double dose group,the mecobalamin treated for two months group and the mecobalamin double dose treated for two months group received mecobalamin treatments accordingly. The morphological changes in the retinal ganglion cells(RGCs) and optic nerve axons were observed and quantified in the specimens.Results:The axons and RGCs in the Vit B 12-treated group were almost the same in comparison to the injury group,while the mecobalamin-treated group has fewer abnormal changes. Results of the quanitification of the axons and RGCs were:354.67±44.72,44.00±8.12,P=0.232,0.170 for the Vit B 12-treated group and 452.17±83.58,58.00±6.38,P=0.010,0.003 for the mecobalamin-treated group. Compared to Vit B 12,mecobalamin had better results(P=0.035,0.037,respectively). The mecobalamin double dose group,the mecobalamin treated for two months group and the mecobalamin double dose treated for two months group had higher numbers than the mecobalamin-treated group.Conclusion:Mecobalamin may have a neuroprotective effect on rat optic nerve injury from crushing and prevents RGCs and axons from degenerating.<
Published: September 30, 2004
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